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Changes in fat homeostasis were linked to increased expression of genes involved in enterocytic triglyceride metabolism and changes in growth with decreased insulin-like growth factor-1 expression. Thus, TIS7 overexpression in the intestine altered growth, metabolic rate, adiposity, and intestinal triglyceride absorption. Sigma-Aldrich offers abstracts and full-text articles by [Yuan Wang, Hristo Iordanov, Elzbieta A Swietlicki, Lihua Wang, Christine Fritsch, Trey Coleman, Clay F Semenkovich, Marc S Levin, Deborah C Rubin]. villus height and crypt depth, resulting in augmented intestinal nutrient absorptive capacity. Previous studies showed that expres abundant overexpression of TIS7 into small intestinal upper crypt and villus enterocytes. TIS7 transgenic mice had increased total. Targeted Intestinal Overexpression of the Immediate Early Gene tis7 in Transgenic Mice Increases Triglyceride Absorption and Adiposity.

Targeted Intestinal Overexpression of the Immediate Early Gene tis7 in Transgenic Mice Increases Triglyceride Absorption and Adiposity Yuan Wang ‡, 1, Hristo Iordanov ‡, 1. 01/11/2018 · CD98 has been implicated in the experimental model of inflammatory bowel disease. We have previously shown that IEC-specific overexpression of CD98 mediates intestinal inflammation and intestinal epithelial barrier dysfunction. Mice overexpressing CD98 exhibited severe colitis and a greater susceptibility to CAC. Here we demonstrated. Overexpression of Intestinal Trefoil Factor in Human Colon Carcinoma Cells Reduces Cellular Growth In Vitro and In Vivo HIROFUMI UCHINO,‡ HIROAKI. 01/11/2018 · CD98 has been implicated in the experimental model of inflammatory bowel disease. We have previously shown that IEC-specific overexpression of CD98 mediates intestinal inflammation and intestinal epithelial barrier dysfunction. Mice overexpressing CD98 exhibited severe colitis and a greater susceptibility to CAC.

Intestinal epithelial cell-specific overexpression of CD98 markedly increased both develop-ment and progress of intestinal adenomas in ApcMin/ mice. Enhanced tumor multiplicity and greater size in CD98 Tg-APCMin/ animals correlated with a rise in proliferation and a fall in apoptosis of intestinal. 13/03/2015 · Autophagy is an intracellular degradation pathway and is considered to be an essential cell survival mechanism. Defects in autophagy are implicated in many pathological processes, including inflammatory bowel disease. Among the innate defense mechanisms of intestinal mucosa, a. Specific overexpression of IL-7 in the intestinal mucosa: the role in intestinal intraepithelial lymphocyte development Hua Yang,1,2 Blair Madison,3 Deborah L..

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